Weight Loss & Metabolic

Tesofensine

Investigational small-molecule triple monoamine reuptake inhibitor (serotonin, norepinephrine, dopamine) originally developed by NeuroSearch, researched for appetite suppression and weight management.

Also known as: NS2330
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What Is Tesofensine

Mechanism & research

Last Updated: July 2026

Tesofensine (development code NS2330) is a small-molecule triple monoamine reuptake inhibitor of the phenyltropane family that blocks presynaptic reuptake of serotonin, norepinephrine, and dopamine, enhancing signaling across all three. It was originally developed by the Danish company NeuroSearch (rights later transferred to Saniona) and first investigated for Alzheimer's and Parkinson's disease; when trials showed pronounced unintended weight loss, it was repurposed as an anti-obesity candidate. It is not a peptide — chemically it is a small molecule (C17H23Cl2NO, 328.28 g/mol) — and it is not approved by the FDA or EMA for any indication; it is supplied for research use only. The lab test table and shop directory above show Peptigrity's independent verification data, updated continuously as new HPLC tests are submitted.

What is Tesofensine and why does the triple-monoamine mechanism matter?

Tesofensine simultaneously inhibits reuptake of all three major monoamines — serotonin, norepinephrine, and dopamine — which is thought to suppress appetite and modestly increase energy expenditure through combined central pathways. This is a fundamentally different mechanism from the GLP-1 class: where semaglutide and retatrutide act on gut-hormone receptors, tesofensine acts directly on brain neurotransmitter systems that regulate satiety and reward — closer in principle to older centrally-acting agents, but hitting three transporters at once. PET imaging in humans has shown dose-dependent dopamine-transporter blockade, and the compound has a notably long elimination half-life (on the order of ~220 hours).

What does the clinical evidence show?

In the landmark 2008 Phase II randomized controlled trial published in The Lancet (Astrup et al.), tesofensine at 0.5 mg produced roughly 10% placebo-subtracted weight loss over 24 weeks — about double the effect of the anti-obesity drugs marketed at the time. This makes tesofensine unusual among research-market compounds in having a published human RCT rather than only preclinical data. However, the data are now well over a decade old, and development did not progress to approval: a cardiovascular safety signal (below) stalled US progress, and the most advanced regulatory action to date is a combination product approved outside the US (Tesomet, tesofensine plus the beta-blocker metoprolol). Evidence level: Human RCT (Phase II); older data, development stalled, no approval.

Is Tesofensine a peptide?

No — Tesofensine is a small-molecule phenyltropane (C17H23Cl2NO, 328.28 g/mol), not a peptide. It is grouped on Peptigrity with peptides because it is sold in the same research-compound market and targets weight management, not because of any structural similarity. The relevant purity signals are therefore HPLC purity and mass-spectrometry identity confirmation against an expected molecular weight of 328.28 Da — the same verification logic we apply to peptides, on different chemistry.

What are the documented side effects of Tesofensine?

The central safety concern is cardiovascular: Phase II trials documented dose-dependent increases in heart rate and blood pressure, and this signal is the main reason US development did not advance. Other reported effects are consistent with monoaminergic stimulation — dry mouth, insomnia, nausea, constipation, and mood or agitation effects. Notably, the Tesomet combination product pairs tesofensine with a beta-blocker specifically to counter the cardiovascular signal through co-administration rather than removing it. Evidence level: Human RCT (Phase II). This is not a self-administration recommendation — see regulatory status below.

What is the regulatory status of Tesofensine?

Tesofensine is not approved by the FDA or EMA; as of 2026 it has no active US approval pathway and is not available by prescription in the United States. The furthest-advanced regulatory action is the approval of the Tesomet combination (with metoprolol) in another market. Development rights passed from NeuroSearch to Saniona. Elsewhere it circulates as a research-use-only compound with no approved prescription pathway. Country-specific import rules apply — see our peptide regulatory status by country guide.

Why does vendor verification matter for Tesofensine?

Tesofensine is active at sub-milligram doses — trial doses ranged from roughly 0.25 to 1 mg — so even small labeling or purity errors translate into large relative dosing errors, which makes independent HPLC and mass-spec testing especially important. The gray market sells it as capsules, tablets, and oral solution, where underdosing, overdosing, and contamination are all real risks. Expected identity on analysis: C17H23Cl2NO, 328.28 Da. Learn how to interpret results in our how-to-test-peptides hub and the reading peptide lab test results guide.

How does Peptigrity verify Tesofensine vendors?

Peptigrity purchases Tesofensine anonymously at standard customer pricing, sends it to independent ISO-accredited labs for HPLC and mass-spectrometry analysis, and publishes the unedited certificate of analysis. The lab test table above shows the full cross-shop dataset, sortable by date, purity, and labeled-vs-tested quantity. Our trust score weights independent lab purity and community reviews equally (50/50); the full methodology is documented in how we calculate trust scores.

This section is for educational and informational purposes only and does not constitute medical advice. Tesofensine is an investigational compound not approved by the FDA or any other regulator for human use. Always consult a qualified healthcare provider before using any research compound. Peptigrity is an independent review platform and does not sell, endorse, or recommend specific products or vendors.

Tesofensine vs other peptides

Same category · Lab-verified
Compound
Mechanism
Avg purity
Tests
Compare
Tesofensine→ This page
Triple monoamine reuptake inhibitor (5-HT/NE/DA)
-
0
GLP-1 / GIP / Glucagon triple-agonist
99.67%
1063
GLP-1 / GIP dual-agonist
99.75%
891
Mitochondrial-derived metabolic peptide
99.43%
458
GLP-1 receptor agonist
99.67%
406
Long-acting amylin receptor agonist
99.62%
239
Compared on independent lab purityBrowse all Weight Loss & Metabolic

Frequently asked

About Tesofensine
How can I verify a Tesofensine vendor is selling real product?
The only reliable purity signal is an independent third-party HPLC certificate of analysis from a lab the vendor does not own or pay — not a COA on the vendor's own letterhead. Peptigrity publishes independent Tesofensine results from ISO-accredited labs in the table above.
Is Tesofensine legal to purchase?
In most jurisdictions Tesofensine can be purchased as a research chemical for laboratory use, but it is not approved for human consumption in the US or EU. Buyers are responsible for verifying local import rules.
Is Tesofensine FDA-approved?
No. Tesofensine is not approved by the FDA or EMA and is not available by prescription in the United States. A combination product (Tesomet, with metoprolol) has been approved in another market, but the single-agent compound has no US or EU approval, largely because of a cardiovascular safety signal seen in Phase II.
Is Tesofensine a peptide?
No — it is a small-molecule phenyltropane, not a peptide. It appears on Peptigrity only because it is sold in the same research-compound market as peptides and targets weight management.
Does Peptigrity recommend a Tesofensine dose?
No. Tesofensine is not approved for human consumption, is active at sub-milligram doses, and carries a documented cardiovascular safety signal. Peptigrity is an independent review platform, not a medical authority; anyone considering an investigational compound should consult a licensed physician.
How does Tesofensine compare to GLP-1 drugs and other weight-loss compounds?
Tesofensine is a centrally-acting appetite suppressant that works on brain monoamine systems, a different mechanism from the gut-hormone GLP-1 class (retatrutide, semaglutide, tirzepatide) and the amylin agonist cagrilintide. Its central safety question is cardiovascular rather than gastrointestinal.

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