Cognitive Enhancement & Neuroprotection

Adamax (Davunetide/NAP)

Neuroprotective octapeptide from ADNP researched for microtubule stabilization and cognitive preservation.

Also known as: Davunetide · NAP · AL-108 · NAPVSIPQ
Avg HPLC purity
99.67%
Across 12 tests
Lab tests on file
14
Independent · HPLC
Shops selling
221
Verified vendors

Lab test results

14 tests · AVG 99.67% · Sorted by date
Date
Shop
Purity
Qty labeled → tested
Endotoxins
Lab
View
Jul 12, 2026
99.40%
10mg10.22mg+2.2%
Jun 5, 2026
-
10mg11.98mg+19.8%
Jun 5, 2026
-
40mg40.98mg+2.4%
Jun 5, 2026
99.00%
10mg10.03mg+0.3%
<1.25 EU/ml
May 6, 2026
99.69%
10mg10.49mg+4.9%
Apr 25, 2026
99.99%
10mg11.37mg+13.7%
Mar 16, 2026
99.96%
10mg10.65mg+6.5%
Feb 16, 2026
99.20%
10mg10.34mg+3.4%
Detected but < 0.01 EU/mg
Feb 13, 2026
99.58%
5mg5.14mg+2.8%
Feb 10, 2026
99.77%
10mg10.94mg+9.4%
< 0.05 EU/mL (PASS for both replicates)
Feb 2, 2026
99.69%
10mg10.49mg+4.9%
Showing 12 of 14 testsShow all 14 tests →

Shops selling Adamax (Davunetide/NAP)

221 verified · top 6 shown

What Is Adamax (Davunetide/NAP)

Mechanism & research

Last Updated: May 2026

Adamax (also Davunetide, NAP, NAPVSIPQ) is a synthetic octapeptide derived from a small active region of ADNP (activity-dependent neuroprotective protein), researched for microtubule stabilization, neuroprotection, and cognitive preservation in neurodegenerative contexts. It is one of the most clinically-developed neuroprotective peptides, having advanced to Phase 2/3 clinical trials for progressive supranuclear palsy (PSP) and mild cognitive impairment, though those trials produced disappointing efficacy results. Davunetide is not approved for any indication and remains in research contexts.

What is Adamax (Davunetide/NAP) and how does the microtubule-stabilizing mechanism work?

Adamax (Davunetide, NAP) is a synthetic octapeptide (Asn-Ala-Pro-Val-Ser-Ile-Pro-Gln) corresponding to a small active region of ADNP (activity-dependent neuroprotective protein), proposed to stabilize neuronal microtubules and protect against the microtubule disruption associated with tau-pathology and other neurodegenerative processes. The microtubule mechanism is mechanistically relevant to tauopathies including Alzheimer's disease, frontotemporal dementia, and progressive supranuclear palsy. Evidence level: Phase 2/3 human RCT (PSP — primary endpoint not met); preclinical extensive (microtubule, neuroprotection).

What does the research show for Adamax/Davunetide?

Davunetide advanced to a Phase 2/3 trial for progressive supranuclear palsy (PSP) by Allon Therapeutics — the trial failed to meet its primary efficacy endpoint, leading to program discontinuation. Earlier Phase 2 work in mild cognitive impairment also produced disappointing efficacy results. The Phase 2/3 failures are an important context: despite mechanistically interesting microtubule-stabilization activity in preclinical work, human efficacy for the studied indications has not been established. Evidence level: Phase 2/3 RCT (failed primary endpoints); preclinical mechanism extensive.

How does Adamax compare to other neuroprotective research peptides?

Adamax has the most advanced clinical development history of any neuroprotective research peptide on Peptigrity, but the Phase 2/3 efficacy failures are the dominant editorial point — preclinical mechanism does not always translate to human clinical benefit.

CompoundMechanismClinical stageOutcome
Adamax (Davunetide/NAP)Microtubule stabilizationPhase 2/3 (PSP)Primary endpoint failed
P21CNTF-derived, neurogenesisPreclinicalMechanism only
SemaxACTH(4-10), BDNFRussian clinical approvedRussia-approved (different mechanism)

What are the documented side effects of Adamax/Davunetide?

The Phase 2/3 PSP trial provided substantial human safety data — common side effects were mild (injection-site reactions, occasional transient headache) with no significant serious adverse event signals. The favorable safety profile is among the more characterized for any research peptide in this category, even though efficacy in the trial was not demonstrated. Evidence level: Phase 2/3 human safety data (substantial); long-term safety beyond trial period less characterized.

What is the regulatory status of Adamax/Davunetide?

Davunetide is not approved for any medical indication anywhere in the world as of May 2026. The Phase 2/3 PSP program by Allon Therapeutics was discontinued after the primary efficacy endpoint was not met. No subsequent sponsor has resumed clinical development. The compound remains available as research-use-only material. Regulatory context in are peptides legal regulatory status by country.

Why does Adamax vendor verification matter for a Phase-3-failed compound?

Because the Phase 2/3 failure means buyers should approach the compound with appropriate skepticism — the mechanism remains interesting but human efficacy for the studied indications has not been established. Vendor verification at least ensures the molecule is what is claimed. Independent HPLC purity testing and mass spectrometry identity confirmation are the standard quality signals. Guidance in how-to-test-peptides hub.

How does Peptigrity verify Adamax vendors?

Peptigrity purchases Adamax vials anonymously at standard customer pricing, sends them to ISO-accredited third-party labs for HPLC and mass spectrometry analysis, and publishes the unedited certificate of analysis. Methodology in how we calculate trust scores.

Frequently Asked Questions

What does Adamax/Davunetide do?

Adamax (Davunetide, NAP) is researched for microtubule stabilization and neuroprotection through a mechanism relevant to tauopathies including progressive supranuclear palsy. A Phase 2/3 trial in PSP failed to meet its primary efficacy endpoint, leading to program discontinuation.

Did the Phase 3 trial of Davunetide succeed?

No. The Phase 2/3 trial of Davunetide in progressive supranuclear palsy (conducted by Allon Therapeutics) failed to meet its primary efficacy endpoint despite favorable safety data. This is an important context for evaluating the compound — preclinical mechanism interest did not translate to human clinical benefit in the studied indication.

Is Adamax legal to purchase?

Adamax (Davunetide) is legal to purchase as a research chemical for laboratory use in most jurisdictions, but it is not approved for human consumption. Country breakdown in peptide legal status guide.

Will Adamax/Davunetide be FDA-approved?

No active clinical development program exists for Davunetide as of May 2026. The Allon Therapeutics program was discontinued after Phase 2/3 failure. FDA approval is unlikely without renewed development by a pharmaceutical sponsor.

How can I verify an Adamax vendor is selling real product?

Independent third-party HPLC certificate of analysis from a lab the vendor does not own or pay, with mass spectrometry identity confirmation. COA interpretation in red flags in peptide certificates of analysis.

Does Peptigrity recommend an Adamax dose?

No. Adamax is not approved for human consumption, has failed Phase 2/3 efficacy for its lead indication, and Peptigrity is an independent review platform, not a medical authority. Anyone considering use should consult a licensed physician and be aware of the failed clinical trial context.

This section is for educational and informational purposes only and does not constitute medical advice. Adamax (Davunetide/NAP) is not approved by the FDA or any other regulator for human use; its Phase 2/3 trial in progressive supranuclear palsy did not meet its primary efficacy endpoint. Always consult a qualified healthcare provider before using any peptide or research compound. Peptigrity is an independent review platform and does not sell, endorse, or recommend specific products or vendors.

Adamax (Davunetide/NAP) vs other peptides

Same category · Lab-verified
Compound
Mechanism
Avg purity
Tests
Compare
Adamax (Davunetide/NAP)→ This page
ADNP-derived microtubule-stabilizing octapeptide
99.67%
14
Tuftsin-derived anxiolytic heptapeptide
99.62%
277
Synthetic ACTH(4-10) heptapeptide analog
99.44%
266
Nine-amino-acid sleep-regulating neuropeptide
99.26%
200
Nonapeptide neurohypophyseal hormone
99.02%
47
Acetylated/amidated SEMAX analog
99.65%
27

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